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Antibiotics including rifaximin and metronidazole have been investigated for their capacity to inhibit TMAO formation by targeting gut bacteria that create TMA

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A number of mechanisms appear to contribute to the mucolytic and mucoregulatory benefits of NAC, including directly breaking disulphide bonds between the molecules forming the mucus with its thiol groups and reducing mucus production through its antioxidant effects (oxidative stress has been shown to increase mucus production)

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