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Metformin inhibits gluconeogenesis from redox-dependent substrates such as lactate and glycerol by targeting mitochondrial glycerol-3-phosphate dehydrogenase (mGPD2), increasing the mitochondrial NADH/NAD + ratio and disrupting the glycerol-3-phosphate shuttle (Figure 2) ( FIGURE 2 Together, these redox and lysosomal pathways allow metformin to suppress hepatic glucose production without broadly impairing mitochondrial respiration
10.1002/hep.28773 Hepatology 20 ChenS.XiaX
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