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And only one, and I repeat, only one was done in humans
Accordingly, AhR activation with various canonical ligands (ITE (5 M), TCDD (10 nM), BaP (5 M), FICZ (5 M), and Indirubin (5 M)) in SKR cells (24 h) induced significantly ACSL4 and PLIN3 protein expression (Appendix Fig S3A)
This condition responds similarly to greater trochanteric pain syndrome given the analogous tendon pathology involved
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It binds to G-actin, which speeds up cell migration throughout the entire body
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