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The rationale for triple agonism is based on preclinical research demonstrating that glucagon receptor activation contributes to energy expenditure and lipid metabolism through distinct mechanisms from those engaged by GLP-1R and GIPR agonism
One reason is that by reducing appetite, these drugs may help individuals develop healthier eating habits, such as reduced portion sizes or more nutritionally-balanced meals, and these habits may persist even after treatment is discontinued
Leukotrienes are a class of classical pro-inflammatory lipid metabolism intermediates that trigger and amplify inflammatory responses (27)
Dietary changes such as LFD could affect the exposure of L-cells to these metabolites, which in turn could affect the production of GLP-1
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The role of tumor necrosis factor-alpha in the pathogenesis and treatment of nonalcoholic fatty liver disease
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