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Enhanced levels of pathogenic bacteria, including , have detrimental effects such as the increased ability of the immune system to promote the deposition of inflammatory and oftentimes pathogenic immunoglobulins in affected organs

Funding This work was supported by grants U24CA270823, U24CA271079, U24CA271012, U24CA271075, U24CA271076, U24CA271037, U24CA271114, U01CA271412, U01CA271410, U01CA271407, U01CA271402 and U24CA210955 from the National Cancer Institutes CPTAC

Receptor-specific differences include D9R7.35b and D15R299 salt bridges shared by GLP-1R and GIPR (but absent in GCGR, which has Q374 in ECL3 and I194 at position 2.67b), and GCGR-specific -stacking between Phe6 and Y138 1.36b