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Many abnormal molecular pathways are involved in these pathophysiological changes, including the abnormal activation of Sirtuin1, Klotho, fibroblast growth factor 21 (FGF21), the renin-angiotensin-aldosterone system (RAAS), Insulin-like Growth Factor Binding Protein (IGFBP), NRF2, and mTOR [120,121]
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"Glucagonocentric restructuring of diabetes: a pathophysiologic and therapeutic makeover"
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doi: 10.1111/j.1464-5491.2006.01943.x 31 LindekildeNScheuerSHRuttersFKnudsenLLasgaardMRubinKHet al
