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This dual engagement may explain why BPC-157 appears to promote not just collagen deposition but organised collagen deposition: the FAK-paxillin axis coordinates cytoskeletal tension, and cells under appropriate cytoskeletal tension deposit collagen with directional preference aligned to the local force field

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Patients may read about thymosin beta-4 wound-healing studies and assume the same conclusions apply directly to commercially marketed TB-500 products

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Interestingly, despite increased expression of the anti-apoptotic protein BCL2 in failing hearts, programmed cell death still occurs, suggesting a complex regulatory process [116]

Equal proportions reflect the equal weight of the two mechanisms in the conceptual framework