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Lipid peroxidation preferentially accumulates at lysosomal membranes and precedes lysosome rupture, transcription factor EB (TFEB) activation, and cathepsin-dependent plasma membrane breakdown, thereby positioning lysosomal stress at the center of population-scale death amplification
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Nevertheless, it is now well-established that all cells can produce persulfides and polysulfides endogenously, through several enzymatic pathways and mechanisms, Cys being the ultimate source of sulfur 14
Tesofensine is generally considered more potent for direct appetite suppression studies due to its neurological mechanism of action
Addition of the ISR agonist Sephin1 to cultured human OLmaintained under control conditions or exposed to ongoing metabolic stressreduced process outgrowth 42 , suggesting that the ISR regulates the protein synthesis machinery required for OL process outgrowth and maintenance
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