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However, liver enlargement and regeneration activated by PXR cause YAP to undergo deacetylation and K63-linked ubiquitination in a Sirt2-dependent manner, which allows YAP-TEAD binding in the nucleus

The possible transport mechanisms together with the wide tissue distribution underlie the role of OCTN2 as the major transporter responsible for the traffic of carnitine and carnitine derivatives in the intestine, distribution to several body districts and reabsorption/excretion in the kidney (Figure 2)

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