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As a follow-up to Lehninger's work several years later, Nishikimi and co-workers observed that the gene that codes for gulonolactone oxidase is actually present in humans, but is not active due to the accumulation of several mutations that turned it into a non-functional pseudogene (Nishikimi & Yagi 1991)
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As a result of the lack of standardized protocols for developing and assessing asthma models, the lack of high-quality, multi-center studies with large sample sizes is due to the limited size and scope of clinical samples
