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AOD9604 appears to stimulate proteoglycan and collagen synthesis in chondrocytes through growth factor signaling pathways distinct from its lipolytic mechanism, supporting cartilage matrix repair in joint models

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One type of mutation, found almost exclusively in people of Ashkenazi Jewish heritage, affects the autonomic and sensory nervous system development causing familial dysautonomia (FD ) Another mutation causes an increased level of phenylalanine (Phe) in the bloodstream resulting in a metabolic disorder called phenylketonuria (PKU)

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