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Chronic neuroinflammation is observed at relatively early stages of disease

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The mechanism of action of DSePA against RP involved its ability to suppress the radiation-induced sustained inflammatory response by upregulating the pulmonary activity level of GPx and concurrently blocking the NF-kB/IL-17/G-CSF/neutrophil axis (Table 2) (Fig

Some reviews describe a broad range of experimental findings, but there remains a gap between promising research and established, FDA-approved clinical use

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