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The pronounced reduction in IL-6, IL-17, and TNF- by QCN-HS is primarily attributable to enhanced intracellular delivery of QCN to key immune and endothelial cells at the wound site, facilitated by the hyaluosome nanocarrier

The data showed that the same concentration of LCS3 mentioned above did not affect the activity of TXNRD1 in VRK2-KO cells, but a higher concentration of LCS3 was required to induce a decline in the activity of TXNRD1 (Fig

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