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The active site of the enzyme that binds glycine to the dipeptide -L-glutamyl-L-cysteine is highly specific (57)
The mechanism is consistent with general lymphoid tissue growth under elevated IGF-1 exposure
Peptides including Tesamorelin, AOD-9604, Tesofensine, FTPP, and MOTS-c are highlighted, and their distinct mechanismssuch as promoting lipolysis, enhancing metabolic efficiency, and controlling appetiteare described
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Upregulation of UHRF1 promotes PINK1-mediated mitophagy to alleviates ferroptosis in diabetic nephropathy
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