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BCAT2s strong correlation with both GPX4 and ACSL4 further supports a link between amino acid metabolism and lipid peroxidation, highlighting the role of metabolic reprogramming
[8] Those data are important for target biology in neuropathic-pain systems, but they do not establish LanCL1 as the mediator of adipocyte lipolysis, fat oxidation, or body-weight effects
The principle is exactly the same
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