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Sustained depletion of NRF2 upon Gclc deletion induces a sex-dependent requirement for NRF2 to maintain liver homeostasis and animal survival Liver-specific deletion of Gclc impaired the expression of lipogenic enzymes and lowered circulating triglyceride levels

Note also that coordination involves loss of protons from the ligand, so the observed mass of the complex is not simply the peptide mass plus the copper mass

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