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However, the therapeutic utility of doxorubicin is undermined by its dose-dependent cardiotoxicity, possibly arising as a result of futile redox cycling, with NADH dehydrogenase within the mitochondria proposed as the most likely site of anthracycline reduction 9,10

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This constant state of poor sleep increases the body's overall stress load and can lead to cortisol dysregulation, which further compounds exhaustion and perpetuates the fatigue cycle

doi: 10.1016/B978-0-443-29044-2.00013-8 69 BhanwalaNKatiyarRKumarSDatusaliaAKKhatikGL