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IVIG immunotherapy protects against synaptic dysfunction in Alzheimers disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling pathway

however, the convalescent and PCC groups displayed minimal separation, indicating limited differences in their protein expression profiles

Pharmacokinetic parameters were evaluated using the WinNonlin software (version 5.3) according to a non-atrioventricular model

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