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However, most current studies rely solely on the EAE mouse model and the LPC-induced localized myelin loss model, which do not fully recapitulate the complex pathological features of MS, particularly its heterogeneity, disease progression, and clinical symptoms

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At the same time, GLP-1 receptor activation reduces the activity of pathogenic Th1 and Th17 cells, the effector T cells most directly responsible for autoimmune tissue damage

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