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The question: does sustained elevation of intracellular NAD+ via NNMT inhibition produce the same long-term benefits as NAD+ precursor supplementation (like nicotinamide riboside or NMN), or are there trade-offs we haven't identified yet

The dose-dependent activation of the FAK-paxillin pathway was confirmed as a key mechanism

Current thoughts on the use of metformin for longevity [1:21:00]

How It Works BPC-157 (Body Protection Compound-157) is a gastric pentadecapeptide that promotes tissue healing through multiple mechanisms: upregulation of growth factor signaling (VEGF, FGF), promotion of angiogenesis (new blood vessel formation), and activation of cellular repair pathways

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