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It has been found that DPP-4/CD26 participates in non-specific inflammation by predominantly regulating the activation and chemotaxis of mononuclear/macrophages, NK cells, and T cells

This additional analysis used data from a trial in subjects with T2D (NCT02877355) [21], which was not included in the main model because not all dosing was supervised

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As expected, Dox treatment elicited ER stress in cells transduced with Ad-TetOn-NHK, but not in cells expressing Ad-GFP

Front Nutr 9:1048258