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4e, PCA analysis indicated that principal component 1 (PC1) covered most of the variation in gene expression (58% of total variance), likely including the effect caused by Gclc deletion
Clinical research, though limited, shows promising results, particularly in alcohol and nicotine use disorders
Individual differences in genetic variants of the thiamine-dependent proteins, in combination with environmental factors, may underlie personal vulnerabilities to thiamine deficiency, manifesting in an ever-increasing variety of clinical presentations
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