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TRIM37 and TRIM28 act with AP2 to remodel chromatin and repress somatic gene programs To further explore the molecular mechanisms underlying the repression of gene expression by the TRIM37TRIM28 complex, we first explored the relationship between chromatin accessibility and gene transcription following Trim37 inactivation using RNA-seq and ATAC-seq analyzes of PGCs at E9.25 (Supplementary information, Fig
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FOXO-1 also increases CD36 expression, enhancing lipid uptake and shifting energy reliance from glucose to lipids
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While concerns about pancreatic safety exist, the overall risk appears to be minimal, and the health risks of having obesity justify the use of GLP-1s
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