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doi: 10.1016/j.neuropharm.2011.12.022 83 Llewellyn-SmithIJReimannFGribbleFMTrappS
Journal of Pediatric Gastroenterology and Nutrition 41:58459
In line with our short-term metabolic analyses, previous studies in New Zealand obese and diet-induced obese male mice suggested that GLP1/E acts primarily at central levels to reverse metabolic alterations by decreasing food intake and body weight, while the potential beneficial effects on pancreatic beta-cells were possibly secondary and indirectly-mediated 33,48
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