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Extracellular matrix components and inflammatory cytokine expression
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Under basal conditions, NRF2 is kept inactive through binding to its negative regulator KEAP1, which is a redox-regulated substrate adaptor for the Cullin (Cul)3-RING-box protein (Rbx)1 ubiquitin ligase complex that directs NRF2 for degradation [10]
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The effect is dose-dependent, most pronounced during titration, and typically improves within 12-16 weeks as the gut adapts to slower transit times
