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A single exposure of doxorubicin between 0 and 50 M, yields minimal cytosolic GSH depletion (2.5% maximum decrease), which recovers within the prescribed time-span (Fig

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These observations imply that ROS function not simply as indicators of oxidative stress during treatment, but also may converge on specific signaling nodessuch as DNA damage response pathways, kinase networks, and mitochondrial remodelingto stabilize drug-resistant cellular states ( Figure 3 )

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