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After TBI, activation of microglia and the cytokines released by damaged cells destroy tight junctions through impairment of BBB endothelial cells, pericytes, astrocytes, and through the synthesis of tight junction proteins
[1] By normalising homocysteine levels, B12 supplementation may reduce this toxic burden and support nerve recovery
IUPAC-IUB Joint Commission on Biochemical Nomenclature (JCBN), and Nomenclature Commission of IUB (NC-IUB), Newsletter
White-fat futile calcium cycling chronic GIPR engagement on white adipocytes triggers SERCA-mediated ER calcium leak
10.1007/s00709-015-0780-y Protoplasma 37 KitaevaA
Injectable peptides are the standard option because they have greater bioavailability and efficacy than oral treatments
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