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After GSH donates its electron, it becomes oxidized and leads to the formation of glutathione disulfide (GSSG)

for 10 days), group III (PTX): amikacin-induced kidney injured mice treated with PTX (50 mg/kg/day) [39,40,41], group IV (CSNPs): amikacin-induced kidney injured mice treated with CSNPs (50 mg/kg/day) [42] and group V (PTX-CSNPs): amikacin-induced kidney injured mice treated with PTX-CSNPs (50 mg/kg/day) [40, 41, 43, 44]

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Wagner MS, Wajner SM, Maia AL

None of the participants were receiving anticoagulant or anti-inflammatory therapy at the time of sampling

The activation of this pathway could imply that BPC-157 plays a role in enhancing the movement and adherence of these cells, which are key processes in tendon healing and regeneration