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It is widely researched for its interaction with intracellular antioxidant-defense systems, oxidative-stress regulation pathways, mitochondrial-support mechanisms, and cellular-maintenance processes
4 In humans and mice, this adaptation is blunted when adenosine is enzymatically degraded 12 or ADORA2B is genetically deleted
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Metabolite concentrations, fluxes and free energies imply efficient enzyme usage
& Pedarzani, P
California Warning: Lead is known to the State of California to cause birth defects or reproductive harm
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