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(1998, Eur J Endocrinol) as a GHS-R1a agonist with GH-release potency comparable to GHRP-6 but without the ACTH, cortisol or prolactin elevation that limits other secretagogues

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The results and data interpretations were again confirmed by electrophoretic mobility shift assays (Fig

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5e) 81 employed template methods (e.g., MgO, CaCO 3 ) combined with KOH activation to prepare porous carbons with ultrahigh SSA (reaching 1980.8 m 2 /g and 1603.4 m 2 /g, respectively)