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These foundational studies defined the receptor pharmacology that underpins all subsequent MT-1 research
It induces metabolic reprogramming in response to hypoxia and growth factor signalling 18, 19 , partly by promoting transcriptional activation of glycolytic genes 20
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Excretion is presumed to occur primarily through renal and hepatic routes, based on typical peptide pharmacokinetics
Endocrinology, Diabetes & Metabolism, 2 (4), e00082
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