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An alternative suggestion is that its metabolite 15oxo-LXA 4 , an electrophilic ,unsaturated ketone, alkylates nucleophilic amino acids and can modulate redox-sensitive and enzyme function, for example to activate erythroid related factor 2 expression and thence anti-inflammatory and repair genes and to inhibit NF-B-regulated pro-inflammatory mediator expression, i.e., the mechanism is not dependent on a specific receptor

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Mitochondrial diseases are characterized by both a diminished capacity to produce ATP as well as an elevated production of reactive oxygen species (ROS) and reactive nitrogen species (RNS) [3]

Uric acid, a peroxynitrite scavenger, inhibits CNS inflammation, blood-CNS barrier permeability changes, and tissue damage in a mouse model of multiple sclerosis

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