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although the pathophysiological mechanism involved in this context is not fully elucidated, whether dependent on the change in the oxidative state, the modification of the immune tolerance system, or the participation of neurokinins.103 In vitiligo, neuropeptide Y is increased in lesional skin and the perilesional area.104 Substance P is increased in the skin of stable vitiligo when compared to unstable disease, suggesting that it is associated with vitiligo stability.105 Extreme physical or psychological stress increases the synthesis of catecholamines and requires the activation of the hypothalamic-pituitary-adrenal axis, which interferes with the immune system regulation

Likewise, researchers examine BPC-157

Excessive ROS surpass the endogenous antioxidant defenses, leading to increased TOS, OSI, and malondialdehyde (MDA), along with decreased TAS 8,9

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Carmeli C, Knyazeva MG, Cunod M, Do KQ

(2024) Key Findings: First-in-human study found intravenous BPC-157 to be safe and well-tolerated in healthy volunteers