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In addition to these mechanisms, oxidative stress activates multiple MAPK pathways, including p38 MAPK, JNK, and ERK1/2

Despite this, a substantial body of pre-clinical and limited clinical research has mapped DSIPs pharmacological effects across multiple systems, identifying several probable mechanistic axes including NMDA receptor modulation, adrenergic system regulation, MAPK/ERK cascade interaction, and mitochondrial respiration effects while acknowledging that the existence of an undiscovered DSIP-like endogenous peptide(s) may account for part of the DSIP-like immunoreactivity observed in tissue distribution studies

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Prietl B, Treiber G, Pieber TR, et al

Glutaminolysis: A hallmark of cancer metabolism