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Together, these peptides represent chemically stable, low-molecular-weight signaling molecules suitable for experimental use in cellular and preclinical research systems

Therapy of supplementation of antioxidants may prevent the risk of CAD in Type II Diabetes

This is the same logic behind loading protocols in many peptide therapies

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The liberated p53 then transcription-independently activates the pro-apoptotic protein BAX, thereby triggering the execution phase of apoptosis