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Because Dihexa promotes structural changes in synaptic architecture rather than temporary neurotransmitter modulation, improvements often persist beyond the treatment period

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These changes contribute to neuroinflammation, bloodbrain barrier (BBB) dysfunction, microglial activation, and neurotransmitter imbalances

11-Hydroxysteroid Dehydrogenase Type 1 (11HSD-1) GH is known to inhibit 11-hydroxysteroid dehydrogenase type 1 (11HSD-1), a microsomal enzyme required for conversion of cortisone to its active metabolite, cortisol, in hepatic and adipose tissue