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It binds actin to influence cell motility and shape, promotes chemotaxis driving cell migration, enhances angiogenesis via cell surface ATP synthase interaction, and reduces inflammation through microRNA-146a upregulation

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Yazinski SA, Comaills V, Buisson R, Genois M-M, Nguyen HD, Ho CK, et al

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Key findings include: In TNBS (trinitrobenzenesulfonic acid) colitis models a standard Crohns disease model BPC-157 administration reduced macroscopic damage scores, histological inflammatory infiltrate, and mucosal ulceration