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While multiple previous studies have confirmed the association between the C4A gene and an increased risk of schizophrenia (132), the intrinsic mechanism underlying the negative correlation between C4B and schizophrenia risk remains unclear
The rate of discontinuation due to adverse events varied from 5% to 19%, predominantly due to gastrointestinal side effects [17]
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Neither compound has been robustly quantified in controlled human appetite studies
Transwell filter migration assays documented enhanced fibroblast migration in dose-dependent patterns
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