faecalis supernatants were capable of disrupting the colonic epithelial monolayer and cleaving GLP-1 in a gelE-dependent manner (Levalley et al., 2020)
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[1] Because gastric mucosal cells are some of the most metabolically active cells in the body, researchers hypothesized that BPC-157 might act as an endogenous cytoprotective signal beyond the gut
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