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In breast cancer cells, a high level of miR-378* induces the metabolic shift from an oxidative to a glycolytic bioenergetics pathway by inhibiting the expression of two PGC-1 partners, ERR (estrogen-related receptor gamma) and GABPA (GA binding protein transcription factor, alpha subunit)
According to Wang et al
Gluck, E
Severe reactions are rare but can occur
doi: 10.1055/s-2007-972854
KPV has been investigated as a research tool in models of intestinal inflammation, including in targeted nanoparticle-based delivery systems (Xiao et al
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