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The damage caused by SAP to the vascular endothelium is manifested as the release of inflammatory mediators and leukocyte adhesion, increased vascular permeability, changes in blood flow shear stress, microthrombosis, and microcirculatory disorders

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By integrating human clinical studies, postmortem analyses, and preclinical models, these findings position KDS2010 as a promising therapy, offering a novel paradigm for PTSD treatment and improving clinical outcomes in PTSD

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