Under conditions of reduced portal insulin-to-glucagon ratio, lipolysis increases in adipose tissue and releases non-esterifed fatty acids which are converted to ketone bodies in the liver through mitochondrial beta oxidation and ketogenesis [25], resulting in a metabolic condition resembling a prolonged fast [26]
Fibre also slows digestion, helping to extend the release of GLP-1 over a longer period, supporting satiety and stable blood sugar levels
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