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Acute striatal necrosis can be divided into three stages [70].The first stage is cytotoxic edema in the basal ganglia, cerebral ischemia, and increased blood volume in the gray matter, which occurs within 24 h after motor deficits, and cranial MRI shows increased signal intensity on T2-weighted imaging of the shell nucleus and caudate nucleus [71], along with diffusion limitation [72]

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By contrast, with continuous administration of GHRP-6, the vagotomized Ghrl / mice exhibited a marked decrease in BT during fasting, although the fluctuation in BT was still observed (Fig