Increasing evidence highlights ferroptosis as a pivotal contributor to the pathogenesis of NDDs (Table 1), suggesting that therapeutic targeting of ferroptosis may offer a promising strategy for the prevention and treatment of NDDs
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Gastroenterology Research: The BPC-157 and TB-500 combination is evaluated for its interactions with gastrointestinal mucosa and systemic tissue-remodeling pathways
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Furthermore, developing small molecule inhibitors targeting BCAA transporters in tumor cells and immunosuppressive cells allows CD8 + T cells to obtain amino acid metabolic advantages in the immune microenvironment and enhance the anti-tumor effect
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