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International Journal of Sports Medicine, 17(5), 320324
In toxin-induced PD models (e.g., MPTP or MPP + ), these inhibitors protect dopaminergic neurons by suppressing iron-mediated ROS formation, reducing lipid peroxidation, and restoring mitochondrial and synaptic integrity (271, 272)
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Metabolomic and fluxomic studies showed that increased CysSSH production by upregulated CSE promotes resistance against BRAF(V600E)-targeted therapy, through protection against drug-induced oxidative damage and feeding the increased cellular energy demand of persister cells
O., Scheible, D., Wijburg, F
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