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Other studies reported that endogenous or exogenous MOTS-c interacts with Nrf2 through direct nuclear entry 60,62
Biomedicine & Pharmacotherapy=Biomdecine & Pharmacothrapie, 57 (34), 145155
View Source Nature Communications MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis 2021 DOI: 10.1038/s41467-020-20790-0 PMID: 33483561 Reynolds JC, Lai RW, Woodhead JST, et al
Whether studied individually for selective pathway investigation or together for dual-mechanism research, both compounds offer significant research utility for programmes in endocrine biology, IGF-1 axis research, body composition science and pituitary function investigation
Breckas team have put thousands of patients on BPC-157 without a reported adverse event (source)
Additionally, the neurogenic locus notch homolog protein 1 (NOTCH1) signalling pathway [133] and the phosphoinositide 3-kinase (PI3K)-protein kinase B (AKT) pathway [176, 177], have also been shown to augment NFE2L2 transcription, and consequently activate NRF2 signalling, thus maintaining redox balance
