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Within the SLNs, macrophages and T-cells were unable to recognize dendritic polymers with carboxyl end groups

The exact cellular mechanisms underlying the synthetic lethality of RSL3 on BRAF/RAS mutant PTC cells remain to be determined

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PMN-MDSCs, on the other hand, facilitate metastasis by restoring the epithelial-to-mesenchymal transition and inducing CSC properties [25]

and patients with glutathione-S-transferase M1 (proinflammatory-cytokine producer macrophages) null and M1 and T1 (Type 1 helper CD4 + lymphocytes) double null polymorphisms had increased mortality suggesting that antioxidant therapy becomes fundamental for those patients (109)