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While caudatin shares mechanistic similarities with compounds such as sulforaphane when modulating downstream NF-B activation, the ability of caudatin to specifically inhibit AP-1 signaling presents a new therapeutic opportunity

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In model experiments employing HEK293 cells in a thiamine-deficient medium, the SLC19A1 variant rs1051266:G exhibits a strong decrease in ThDP efflux compared to the canonic SLC19A1 sequence rs1051266:A