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Compared to mAChRs, nAChRs have a reduced affinity for the natural agonist ACh, which suggests that they may be involved in negative feedback mechanisms linked with growing levels of ACh
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Isoureguanidine alkaloids target the miR-122-5p/TP53/SLC7A11 axis, upregulating miR-122-5p and SLC7A11 while suppressing TP53 to prevent ferroptosis (215)
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