Although pre-clinical and clinical confirmation is required, these studies suggest that PTC may in part be driven by UCA1 acting as a ceRNA to suppress miR-204-5p and stimulate oncogenic IGFBP-5 [70]
In: Handbook of Clinical Neurology, Volume 120, Chapter 61
Thioredoxin mediates oxidation-dependent phosphorylation of CRMP2 and growth cone collapse
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